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Neurotoxicity of Metals

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Attention for Chapter 16: Neurotoxicity of Copper
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Chapter title
Neurotoxicity of Copper
Chapter number 16
Book title
Neurotoxicity of Metals
Published in
Advances in neurobiology, January 2017
DOI 10.1007/978-3-319-60189-2_16
Pubmed ID
Book ISBNs
978-3-31-960188-5, 978-3-31-960189-2

Felix Bulcke, Ralf Dringen, Ivo Florin Scheiber, Bulcke, Felix, Dringen, Ralf, Scheiber, Ivo Florin


Copper is an essential trace metal that is required for several important biological processes, however, an excess of copper can be toxic to cells. Therefore, systemic and cellular copper homeostasis is tightly regulated, but dysregulation of copper homeostasis may occur in disease states, resulting either in copper deficiency or copper overload and toxicity. This chapter will give an overview on the biological roles of copper and of the mechanisms involved in copper uptake, storage, and distribution. In addition, we will describe potential mechanisms of the cellular toxicity of copper and copper oxide nanoparticles. Finally, we will summarize the current knowledge on the connection of copper toxicity with neurodegenerative diseases.

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The data shown below were collected from the profiles of 31 X users who shared this research output. Click here to find out more about how the information was compiled.
Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 74 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 74 100%

Demographic breakdown

Readers by professional status Count As %
Student > Ph. D. Student 13 18%
Student > Bachelor 11 15%
Student > Doctoral Student 6 8%
Researcher 4 5%
Student > Master 4 5%
Other 10 14%
Unknown 26 35%
Readers by discipline Count As %
Pharmacology, Toxicology and Pharmaceutical Science 8 11%
Medicine and Dentistry 8 11%
Neuroscience 6 8%
Chemistry 5 7%
Biochemistry, Genetics and Molecular Biology 5 7%
Other 10 14%
Unknown 32 43%