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C-Myc in B-Cell Neoplasia

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Cover of 'C-Myc in B-Cell Neoplasia'

Table of Contents

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    Book Overview
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    Chapter 1 The c-myc Story: Where we’ve been, Where we seem to be Going
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    Chapter 2 B-lymphocyte-lineage Cells from Early Precursors to Ig-secreting Plasma Cells: Targets of Regulation by the myc/mad/max Families of Genes?
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    Chapter 3 DNA conformation, topology, and the regulation of c-myc expression.
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    Chapter 4 Differential Regulation of the c-MYC P1 and P2 Promoters in the Absence of Functional Tumor Suppressors: Implications for Mechanisms of Deregulated MYC Transcription
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    Chapter 5 Positive and Negative Regulation of c- Myc Transcription
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    Chapter 6 Structure and Expression of the c-Myc/Pvt 1 Megagene Locus
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    Chapter 7 Murine and human 3'IgH regulatory sequences.
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    Chapter 8 Receptor-specific Regulation of NF-κB, c-Myc and Fas-mediated Apoptosis in Primary B Cells
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    Chapter 9 c-myc promotes survival of WEHI 231 B lymphoma cells from apoptosis.
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    Chapter 10 Role of c-myc and p27 in anti-IgM induced B-lymphoma apoptosis.
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    Chapter 11 Mnt: A Novel Max-interacting Protein and Myc Antagonist
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    Chapter 12 Regulation of Cell Growth by the Myc-Max-Mad Network: Role of Mad Proteins and YY1
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    Chapter 13 C-Myc Transrepression and Cell Transformation
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    Chapter 14 Association of Myc with the zinc-finger protein Miz-1 defines a novel pathway for gene regulation by Myc.
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    Chapter 15 Regulated expression and function of the c-Myc antagonist, Mad1, during a molecular switch from proliferation to differentiation.
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    Chapter 16 A biochemical and biological analysis of Myc superfamily interactions.
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    Chapter 17 Design of Dominant Negatives to bHLHZip Proteins that Inhibit DNA Binding
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    Chapter 18 Myc Target Genes in Neoplastic Tranformation
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    Chapter 19 Cytokine-induced Inhibition of Myc Activity in Monocytic Cells
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    Chapter 20 c-Myc Dependent Initiation of Genomic Instability During Neoplastic Transformation
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    Chapter 21 The Role of c -myc During Normal B Cell Proliferation, and as B Cells Undergo Malignant Transformation
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    Chapter 22 Lymphomas and plasmacytomas in transgenic mice involving bcl2, myc and v-abl.
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    Chapter 23 Molecular pathogenesis of myc-initiated B-cell lymphomas in the bursa of Fabricius.
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    Chapter 24 Genetic Recombinations Between c- myc and Ighμ as Precursors for Recombinations Between c-myc and Ighα in BALB/c Plasmacytomas
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    Chapter 25 Generation of Immunoglobulin/c- myc Recombinations in Murine Peyer’s Patch Follicles
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    Chapter 26 Myc/max family of transcription factors and bcl-2 are involved in drug-induced apoptosis of myeloma cells.
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    Chapter 27 Effects of interferon alpha on myeloma cells: mechanisms of differential responsiveness.
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    Chapter 28 Investigation of aberrant translational control of c-myc in cell lines derived from patients with multiple myeloma.
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    Chapter 29 Dysregulation of c-myc in multiple myeloma.
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    Chapter 30 IgH translocations in multiple myeloma: a nearly universal event that rarely involves c-myc.
Attention for Chapter 11: Mnt: A Novel Max-interacting Protein and Myc Antagonist
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Chapter title
Mnt: A Novel Max-interacting Protein and Myc Antagonist
Chapter number 11
Book title
C-Myc in B-Cell Neoplasia
Published by
Springer, Berlin, Heidelberg, January 1997
DOI 10.1007/978-3-642-60801-8_11
Pubmed ID
Book ISBNs
978-3-64-264560-0, 978-3-64-260801-8
Authors

P. J. Hurlin, C. Qúeva, R. N. Eisenman, Hurlin, P. J., Qúeva, C., Eisenman, R. N.

Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 12 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 12 100%

Demographic breakdown

Readers by professional status Count As %
Student > Bachelor 2 17%
Researcher 2 17%
Professor 1 8%
Other 1 8%
Student > Master 1 8%
Other 1 8%
Unknown 4 33%
Readers by discipline Count As %
Biochemistry, Genetics and Molecular Biology 3 25%
Agricultural and Biological Sciences 3 25%
Medicine and Dentistry 1 8%
Unknown 5 42%