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Molecular Mechanisms in the Pathogenesis of Idiopathic Nephrotic Syndrome

Overview of attention for book
Cover of 'Molecular Mechanisms in the Pathogenesis of Idiopathic Nephrotic Syndrome'

Table of Contents

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    Book Overview
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    Chapter 1 History of Research on Pathogenesis of Idiopathic Nephrotic Syndrome
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    Chapter 2 Hemopexin in Minimal Change Nephrotic Syndrome
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    Chapter 3 Angiopoietin-Like 4 (Angptl4) in MCNS
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    Chapter 4 Co-stimulatory Molecule CD80 (B7.1) in MCNS
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    Chapter 5 Energy and Mammalian Target of Rapamycin Complex 1 (mTORC1) in Minimal Change Nephrotic Syndrome
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    Chapter 6 The Role of c-mip in the Pathogenesis of Minimal Change Nephrotic Syndrome
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    Chapter 7 Regulatory T Cells and Oxidative Stress in Minimal Change Nephropathy
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    Chapter 8 Cytokines as Active Factors in Minimal Change Nephrotic Syndrome
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    Chapter 9 Soluble Urokinase-Type Plasminogen Activator Receptor (suPAR) in Focal Segmental Glomerulosclerosis
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    Chapter 10 Cytokines as Active Factors in Focal Segmental Glomerulosclerosis
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    Chapter 11 M-type Phospholipase A2 Receptor (PLA2R) and Thrombospondin Type-1 Domain-Containing 7A (THSD7A) in Membranous Nephropathy
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    Chapter 12 Cationic Bovine Serum Albumin as Cause of Membranous Nephropathy: From Mice to Men
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    Chapter 13 Podocytes as a Direct Target of Drugs Used in Idiopathic Nephrotic Syndrome
Attention for Chapter 2: Hemopexin in Minimal Change Nephrotic Syndrome
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Citations

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Chapter title
Hemopexin in Minimal Change Nephrotic Syndrome
Chapter number 2
Book title
Molecular Mechanisms in the Pathogenesis of Idiopathic Nephrotic Syndrome
Published by
Springer, Tokyo, January 2016
DOI 10.1007/978-4-431-55270-3_2
Book ISBNs
978-4-43-155269-7, 978-4-43-155270-3
Authors

Yasuko Kobayashi, Moin A. Saleem, Kobayashi, Yasuko, Saleem, Moin A.

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Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 1 Mendeley reader of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 1 100%

Demographic breakdown

Readers by professional status Count As %
Unknown 1 100%
Readers by discipline Count As %
Medicine and Dentistry 1 100%

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