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Damage-Associated Molecular Patterns in Human Diseases

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Cover of 'Damage-Associated Molecular Patterns in Human Diseases'

Table of Contents

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    Book Overview
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    Chapter 1 Prologue: The Term “Immunity” over the Course of Time
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    Chapter 2 The Three Major Paradigms in Immunology
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    Chapter 3 Epilogue: What Major Paradigm in Immunology Comes Next?
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    Chapter 4 Prologue: The Guardians of Homeostasis
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    Chapter 5 Innate Immune Recognition Molecules
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    Chapter 6 Epilogue: The Innate Immune System: A Global Player in Health and Disease
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    Chapter 7 Prologue: The Whole Body as an Immune System
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    Chapter 8 Mobile Innate Immune Cells
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    Chapter 9 Sessile Innate Immune Cells
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    Chapter 10 Epilogue : Cells of Our Immune System: A Whole Army of Defenders Far Beyond T and B Cells
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    Chapter 11 Prologue: About DAMPs, PAMPs, and MAMPs
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    Chapter 12 Endogenous DAMPs, Category I: Constitutively Expressed, Native Molecules (Cat. I DAMPs)
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    Chapter 13 Endogenous DAMPs, Category II: Constitutively Expressed, Injury-Modified Molecules (Cat. II DAMPs)
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    Chapter 14 Endogenous DAMPs, Category III: Inducible DAMPs (Cat. III DAMPs)
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    Chapter 15 Exogenous DAMPs, Category IV (Cat. IV DAMPs)
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    Chapter 16 Epilogue: The Well-Dosed Work of DAMPs in Their Commission to Restore and Maintain Homeostasis
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    Chapter 17 Prologue: Innate Immune Tools to Defend Against Stressful Injury: Success and Failure
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    Chapter 18 Cell-Autonomous (Cell-Intrinsic) Stress Responses
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    Chapter 19 Regulated Cell Death
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    Chapter 20 Epilogue: The Horror of an Injury-Induced Avalanche of DAMPs
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    Chapter 21 Prologue: The Various Facets of Innate Immune Effector Responses
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    Chapter 22 Cellular Inflammatory Responses
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    Chapter 23 Humoral Innate Immune Effector Responses
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    Chapter 24 Regulation of Innate Inflammatory Responses
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    Chapter 25 Epilogue: How Should Clinicians Implement the Complex Innate Immune Effector Responses at the Patient’s Bedside?
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    Chapter 26 Prologue: The “Ready-to-Go” Lymphocytes of the Immune System
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    Chapter 27 Activation and Function of Innate Lymphoid Cells
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    Chapter 28 Activation and Function of Unconventional T Cells
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    Chapter 29 Epilogue: Killer Lymphocytes as Tools for Future Immunotherapeutic Approaches
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    Chapter 30 Prologue: The “Long Arm” of DAMPs in Shaping Adaptive Immune Responses and Tissue Repairing Processes
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    Chapter 31 Antigen Uptake, Processing, and Presentation by Dendritic Cells
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    Chapter 32 Antigen in the Presence of DAMPs Induces Immunostimulatory Dendritic Cells to Promote Destructive Adaptive Immune Responses
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    Chapter 33 Antigen in the Absence of DAMPs Promotes Immune Tolerance: The Role of Dendritic Cells and Regulatory T Cells
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    Chapter 34 An Impressive Example of Peripheral Tolerance Against Nonself: Tolerance to Commensal Bacterial and Dietary Protein Antigens
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    Chapter 35 Immunometabolism of Dendritic Cells and T Cells
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    Chapter 36 Role of DAMPs in Tissue Regeneration and Repair
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    Chapter 37 Epilogue: The “DAMPome” as a Key Player in the Pathogenesis of Many Human Diseases
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Title
Damage-Associated Molecular Patterns in Human Diseases
Published by
Springer International Publishing, June 2018
DOI 10.1007/978-3-319-78655-1
ISBNs
978-3-31-978654-4, 978-3-31-978655-1
Authors

Land, Walter Gottlieb

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The data shown below were collected from the profiles of 2 X users who shared this research output. Click here to find out more about how the information was compiled.
Mendeley readers

Mendeley readers

The data shown below were compiled from readership statistics for 11 Mendeley readers of this research output. Click here to see the associated Mendeley record.

Geographical breakdown

Country Count As %
Unknown 11 100%

Demographic breakdown

Readers by professional status Count As %
Researcher 3 27%
Professor 2 18%
Student > Ph. D. Student 2 18%
Student > Postgraduate 1 9%
Unknown 3 27%
Readers by discipline Count As %
Agricultural and Biological Sciences 2 18%
Immunology and Microbiology 2 18%
Biochemistry, Genetics and Molecular Biology 1 9%
Medicine and Dentistry 1 9%
Neuroscience 1 9%
Other 1 9%
Unknown 3 27%