Chapter title |
Tenascin-C is a possible mediator between initial brain injury and vasospasm-related and -unrelated delayed cerebral ischemia after aneurysmal subarachnoid hemorrhage.
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Chapter number | 20 |
Book title |
Neurovascular Events After Subarachnoid Hemorrhage
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Published in |
Acta neurochirurgica Supplement, November 2014
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DOI | 10.1007/978-3-319-04981-6_20 |
Pubmed ID | |
Book ISBNs |
978-3-31-904980-9, 978-3-31-904981-6
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Authors |
Suzuki H, Kanamaru K, Shiba M, Fujimoto M, Kawakita F, Imanaka-Yoshida K, Yoshida T, Taki W, Hidenori Suzuki, Kenji Kanamaru, Masato Shiba, Masashi Fujimoto, Fumio Kawakita, Kyoko Imanaka-Yoshida, Toshimichi Yoshida, Waro Taki, Suzuki, Hidenori, Kanamaru, Kenji, Shiba, Masato, Fujimoto, Masashi, Kawakita, Fumio, Imanaka-Yoshida, Kyoko, Yoshida, Toshimichi, Taki, Waro |
Abstract |
Tenascin-C (TNC), a matricellular protein, exerts diverse functions, including tissue remodeling and apoptosis, and is induced in cerebrospinal fluid (CSF) after aneurysmal subarachnoid hemorrhage (SAH). The purpose of this study was to examine the relationships among CSF TNC levels, initial brain injury, delayed cerebral ischemia (DCI), and vasospasm after SAH. |
X Demographics
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United States | 1 | 100% |
Demographic breakdown
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Members of the public | 1 | 100% |
Mendeley readers
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Country | Count | As % |
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Unknown | 29 | 100% |
Demographic breakdown
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Student > Bachelor | 7 | 24% |
Student > Ph. D. Student | 4 | 14% |
Other | 3 | 10% |
Researcher | 2 | 7% |
Student > Postgraduate | 2 | 7% |
Other | 6 | 21% |
Unknown | 5 | 17% |
Readers by discipline | Count | As % |
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Medicine and Dentistry | 15 | 52% |
Engineering | 2 | 7% |
Social Sciences | 1 | 3% |
Biochemistry, Genetics and Molecular Biology | 1 | 3% |
Materials Science | 1 | 3% |
Other | 1 | 3% |
Unknown | 8 | 28% |